Root-Cause Healing Approaches for Pain Symptom Management

Dive into the world of pain symptom management and root-cause healing. Find powerful methods to address health challenges at their source.

Abstract

I am Dr. Alexander Jimenez, DC, APRN, FNP-BC. In this educational post, I present a cohesive, evidence-based narrative that reflects the core themes discussed in our recent gathering on transforming healthcare—reframed from my first-person perspective and enriched with my clinical observations and practice insights from Personal Injury Doctor Group. This post integrates modern research methods, critical appraisal of literature from leading investigators, and practical protocols I use daily to improve patient outcomes. The goal is to trace where medicine has been, define where it must go, and detail how each of us can apply integrative, root-cause strategies—anchored in science—to restore health, vitality, and medical freedom.

We begin by examining the “history of the future of medicine”: how centuries of tradition, observation, and emergent science created today’s landscape—one too often dominated by reactive, protocol-only care, third-party constraints, and symptom suppression. I explore why evidence-based care must be more than adherence to static guidelines—why it must include critical thinking, physiological first principles, and tailored plans that respect biochemical individuality, genetics, and epigenetics. I contextualize the rise of pharmaceutical management of chronic disease, the unintended consequences of certain population-wide targets (such as aggressive lipid suppression), and the need to reintegrate foundational pillars like nutrition, physical activity, sleep, stress regulation, hormone balance, and metabolic health into mainstream practice.

Next, I present a comprehensive, system-by-system exploration of root-cause, integrative therapeutics. We will discuss the physiological underpinnings of hormone optimization (including estrogen, progesterone, testosterone, DHEA, and thyroid hormones), nutrition and metabolism (insulin sensitivity, mitochondrial biology, nutrient density, and inflammatory modulation), neuroendocrine-immune crosstalk, and cardiometabolic risk. Throughout, I provide the rationale for each intervention—why it works, when it’s indicated, and how to monitor effectiveness and safety. I incorporate pragmatic frameworks supported by clinical research and echoing what I see in practice: that personalized, multimodal strategies often reverse patterns of chronic illness previously considered inexorable.

I also examine medical freedom and clinical autonomy as quality-of-care issues—why patient choice and practitioner judgment must be preserved, and how we leverage the scientific method to validate therapies such as bioidentical hormone replacement therapy (BHRT), peptide therapeutics, exosome-based approaches (in appropriate research or clinical contexts), targeted supplementation, and structured lifestyle modification. I share the operational side: why adherence matters, how delivery methods (such as BHRT pellets) can improve compliance for select patients, and how to simplify plans without sacrificing sophistication. You will find decision trees and clinical reasoning models that help translate systems biology into daily practice.

Additionally, I weave in concrete patient stories (anonymized, representative composites) from my clinic—nagging migraines resolved by identifying and treating thyroid and perimenopausal shifts; stubborn hypertension improved via insulin sensitization, sleep remediation, and weight-bearing exercise; recurrent musculoskeletal pain quelled by anti-inflammatory nutrition and microvascular conditioning; post-traumatic stress physiology calmed by sleep, HRV-guided breathing, and targeted micronutrients. Each vignette demonstrates the causal logic linking physiology to symptom resolution.

We will also address policy inflection points, from recent regulatory shifts (for example, evolving FDA stances and black box changes affecting estrogen labeling) to proposed nutritional education standards for clinicians, and how these developments align or clash with current evidence. The take-home message: robust, modern evidence supports a proactive, patient-centered model that integrates lifestyle medicine, precision endocrinology, and the ethical use of therapeutics—measured by meaningful outcomes.

Finally, the post closes with a 500-word “Summary,” “Conclusion,” and “Key Insights,” dated to reflect the creation date. These sections distill the clinical frameworks, research highlights, and implementation strategies discussed herein. I hope that this edition of “catio” as a resource e” powers practitioners and patients alike to restore health through sound science, compassionate care, and disciplined, root-cause reasoning.

Keywords at the end provide search relevance; references include landmark trials, systematic reviews, consensus statements, and methodological resources to support evidence-based practice.

Empowered, Personalized Healthcare: The Future Begins with Root Cause

I have long believed that the most meaningful change in healthcare occurs when we choose to address root causes rather than accumulate prescriptions. As a DC and FNP-APRN trained in both manual and advanced medical practice, I see daily how a patient’s biology responds when we correct underlying drivers—insulin resistance, micronutrient depletion, circadian disruption, hormone dysregulation, mitochondrial dysfunction, autonomic imbalance, mechanical loading errors, or chronic inflammation. This is not a theoretical stance. It is a clinical reality, aligned with modern systems biology, and validated by the literature, which goes far beyond symptom-based algorithms.

At Personal Injury Doctor Group, my team and I serve individuals with complex pain syndromes, metabolic derangements, sports injuries, post-crash musculoskeletal dysfunction, post-concussive syndromes, and chronic fatigue. What unites these cases is a shared physiology: inflammation, impaired tissue repair, immune activation, autonomic dysregulation, and often neglected endocrine contributors. When we orchestrate care across these axes—nutrition, hormones, sleep, movement, stress modulation—patients regain not just function but identity. That is the promise of empowered, personalized healthcare.

From Observation to Evidence: The History of the Future of Medicine

• 1700s–1800s: Medicine relied heavily on observation and tradition, with technological tools just beginning to emerge. The scientific method was gaining traction, but was limited by laboratory capabilities.
• Early 1900s: Science and industry accelerated medical advances. Care became more protocol-driven—an achievement for standardization and safety, but one that sometimes calcified innovation.
• Mid–late 20th century: Standard of care took hold as a legal and social framework. While guidelines saved lives, they sometimes discouraged hypothesis testing beyond the consensus, even when backed by emerging data.
• 1980s onward: Pharmaceutical innovation delivered tremendous benefits—antibiotics, antiretrovirals, antihypertensives, and more. Yet the pendulum swung toward pharmacologic symptom control, with insufficient emphasis on nutrition and lifestyle as primary therapeutics for chronic disease.
• 2010s: A confluence of regulatory structures, payer incentives, and corporate consolidation increasingly constrained clinician autonomy and patient choice. Preventive and integrative practices grew, but often at the margins.
• 2020s–present: Evidence for lifestyle-first, hormone-informed, and precision therapeutics has matured. Nutritional science, chronobiology, immunometabolism, and endocrinology offer powerful, non-exclusive complements to pharmacology. Regulatory shifts suggest a slow but meaningful re-centering of preventative and individualized care.

The “future of medicine” is a return to fundamentals—what we eat, how we sleep, how we move, how we recover—augmented by precise bioidentical hormones, metabolically rational medications when needed, and” novel biologics w” with rigorous safety and ethical frameworks. It is science meeting humanity through critical thinking.

Why Protocol-Only Care Falls Short: Physiological Individuality and Critical Thinking

Every patient carries unique genetics, epigenetic marks, microbiota composition, and environmental exposures. Two individuals with identical LDL-C may harbor drastically different atherogenic risk profiles based on LDL particle number (LDL-P), ApoB, insulin resistance, Lp(a), inflammatory markers (hs-CRP), endothelial function, sleep quality, and autonomic tone. Protocols that ignore this complexity risk under- or over-treatment.

Critical thinking means asking:

• What is the primary physiological bottleneck?
• What perturbations in systems biology are maintaining the illness?
• What non-pharmacologic levers can most effectively restore homeostasis?
• Which medications, if any, improve risk-to-benefit when layered atop foundational care?

The answer is rarely one-size-fits-all. It is a personalized algorithm informed by evidence, lab indices, imaging, and the patient’s lived experience.

The Cost of Symptom Suppression: A Deep Dive into Chronic Disease Dynamics

Chronic disease is the downstream expression of dysregulated systems—glucose and lipid metabolism, mitochondrial energy production, autonomic balance, endocrine signaling, and immune surveillance. Symptom suppression—analgesics for pain, sedatives for insomnia, proton pump inhibitors for reflux—may be appropriate short-term but can perpetuate pathology if we never ask “why.”

Consider the migraineur who relies on triptans alone. In my clinic, we often find subclinical hypothyroidism, perimenopausal estrogen volatility, magnesium deficiency, disrupted sleep architecture, and cervical myofascial dysfunction feeding the same loop. When we stabilize hormones (bioidentical estrogen/progesterone, if indicated), replete magnesium and riboflavin, correct sleep, and treat cervical mechanics, the frequency and intensity of attacks can fall dramatically. Triptans remain an option, but no longer the crutch.

Rethinking Lipids: Cholesterol, Brain Health, and Immune Communication

Cholesterol is essential for membrane integrity, myelination, steroidogenesis, and synaptic function. Research continues to refine our understanding of lipid physiology:

• ApoB-containing particles drive atherogenesis; LDL-C alone may not fully describe risk.
• The brain’s cholesterol metabolism is largely autonomous; disruptions can affect cognition.
• Dendritic cells and other immune cells require membrane cholesterol for signaling and antigen presentation; exbrain’s in lipid manipulation may have unintended effects.

The implication is not to abandon lipid-lowering therapy; statins and other agents can be life-saving in appropriate contexts. Rather, we must personalize targets and modalities:

• Evaluate ApoB and LDL-P for atherogenic particle burden.
• Account for insulin resistance, triglyceride/HDL ratio, Lp(a), and inflammatory markers.
• Consider non-statin options for statin intolerance or specific risk patterns (ezetimibe, bempedoic acid, PCSK9 inhibitors).
• Pair pharmacology with lifestyle strategies shown to improve atherogenic dyslipidemia (Mediterranean-pattern nutrition, fiber, resistance training, glycemic control, restorative sleep).

Cognition must be monitored when LDL-C is lowered intensively in select populations. Tailoring therapy, not abandoning it, balances cardioprotection with neurological well-being.

Medical Freedom and Patient Choice: Quality of Care and Ethical Practice

Healthcare quality depends on the clinician’s ability to integrate evidence with patient values and physiological individuality. When insurers or formularies restrict rational, evidence-based options, the result can be suboptimal care. It is simple:

• Preserve clinician autonomy to use best evidence, sound judgment, and patient-informed consent.
• Preserve patient choice to pursue integrative, lifestyle-forward care.
• Hold all therapies—conventional and integrative—to the same scientific standards of safety and efficacy.

In my practice, this means rigorous documentation, baseline labs, risk stratification, progress monitoring, and outcome measurement, regardless of whether I prescribe a statin, bioidentical hormones, a peptide in sanctioned contexts, or a nutrition and exercise protocol.

The Pillars of Personalized, Integrative Care

Nutrition as Foundational Therapy

Nutrition is a primary drug—affecting insulin signaling, inflammation, microbiome composition, endothelial function, and neurochemistry.

Key principles I apply:

• Emphasize minimally processed, nutrient-dense foods: vegetables, fruits, legumes (as tolerated), nuts, seeds, lean proteins, fatty fish, and healthy fats (e.g., olive oil, avocado).
• Align carbohydrates with metabolic status: lower-glycemic-load options for insulin resistance; strategic carbohydrates for athletes or individuals with high energy demands.
• Ensure adequate protein for sarcopenia prevention and tissue repair: generally 1.2–1.6 g/kg/day, adjusted for renal function and clinical context.
• Increase fiber intake (25–40 g/day) to support glycemic control, lipid modulation, and microbiome diversity.
• Identify and eliminate contributors to inflammation and dysbiosis: excessive refined sugars, trans fats, ultra-processed foods, and individual intolerances.

Rationale:

• Lowering insulin resistance reduces hepatic VLDL output, improves HDL function, and decreases the formation of small, dense LDL.
• Omega-3 fatty acids modulate eicosanoid pathways and membrane fluidity, supporting anti-inflammatory signaling.
• Polyphenols and fermentable fibers support short-chain fatty acid production, enhancing gut barrier integrity and immune tolerance.

Movement and Physical Conditioning

Exercise rewires physiology:

• Resistance training increases GLUT4 translocation, insulin sensitivity, and myokine secretion (e.g., IL-6 in the anti-inflammatory context of skeletal muscle).
• Aerobic training improves mitochondrial biogenesis via PGC-1?, enhances endothelial function, and reduces blood pressure.
• High-intensity intervals, when appropriate, improve VO2 max and cardiometabolic resilience.

Protocols I prefer:

• Strength training 2–3 times weekly, prioritizing multi-joint movements.
• Zone 2 aerobic training for mitochondrial efficiency.
• Mobility and neuromuscular training for injury prevention and functional capacity.

Sleep and Circadian Health

Sleep is an endocrine organ unto itself:

• Slow-wave sleep supports growth hormone secretion and anabolic recovery.
• Circadian alignment governs insulin sensitivity and cortisol rhythm.
• Sleep restriction elevates ghrelin, reduces leptin, and increases caloric intake.

Interventions:

• Fixed sleep/wake windows; light exposure in the morning; avoidance of blue light at night.
• Cognitive-behavioral therapy for insomnia (CBT-I) is the first line.
• Magnesium glycinate, sleep hygiene, and, if indicated, short-term pharmacologic aids.

Stress Regulation and Autonomic Balance

Chronic sympathetic dominance impairs insulin sensitivity, elevates blood pressure, and increases inflammatory cytokines.

Tools:

• Heart rate variability (HRV) biofeedback and diaphragmatic breathing.
• Mindfulness-based stress reduction, progressive muscle relaxation.
• Graded exposure and cognitive strategies for those with trauma history.

Hormone Optimization: Evidence-Informed, Patient-Centered

Hormones orchestrate systemic physiology. Correcting clinically significant deficiencies can be transformative.

Estrogen and Progesterone (Bioidentical, When Appropriate)

Evidence supports individualized, carefully monitored BHRT for select women:

• Benefits: vasomotor symptom relief, bone protection, potential cognitive support when initiated near menopause, urogenital health, and improved sleep.
• Risk minimization: transdermal estradiol may have a lower VTE risk than oral estradiol in appropriate patients; micronized progesterone may be better tolerated than synthetic progestins.
• Monitoring: mammography as indicated, endometrial surveillance for abnormal bleeding, BP, lipids, and symptom assessment.

Rationale:

• Estrogen modulates osteoclast activity, enhances nitric oxide synthase in endothelium, and supports synaptic plasticity.
• Progesterone modulates GABAergic tone, improving sleep quality and anxiety in some women.

The regulatory context evolves, and we adapt with a commitment to the best available evidence and patient safety.

Testosterone and DHEA

In women:

• Potential benefits: improved libido, mood, energy, and body composition when used in physiological female ranges.
• Risks: virilization if overdosed, lipid changes; careful dosing and monitoring are essential.

In men:

• Treat clinical hypogonadism with documented low testosterone and symptoms.
• Benefits: improved anemia, bone density, body composition, libido, and depressive symptoms in some.
• Monitor hematocrit, PSA (with shared decision-making), estradiol, and lipids.

Rationale:

• Testosterone modulates protein synthesis, erythropoiesis, mood, and metabolic rate.

Thyroid Optimization

Subclinical hypothyroidism or poor peripheral conversion can present as fatigue, cold intolerance, dyslipidemia, constipation, and cognitive fog.

Approach:

• Evaluate TSH, free T4, free T3, thyroid antibodies, and clinical signs.
• Begin with levothyroxine for overt hypothyroidism; consider combination therapy in select cases after risk-benefit discussion.
• Address nutrient cofactors (selenium, iodine within safe ranges, and iron status).

Rationale:

• Thyroid hormones regulate mitochondrial biogenesis, basal metabolic rate, LDL receptor expression, and mood.

Peptides and Biologics: Science-Guided, Safety-First

Peptide therapeutics (where permitted and supported by evidence) can provide targeted signaling modulation:

• Examples include agents under investigation for support of the GH axis, tissue repair, or metabolic effects.
• Safety and regulatory compliance are paramount: use vetted sources, adhere to jurisdictional regulations, and obtain informed consent.

Exosomes and other biologics remain areas of active research. I approach them within ethical frameworks—clinical trials where possible, meticulous screening, and transparent risk-benefit discussions.

The Case for Simplicity: Adherence Is a Clinical Outcome

A brilliant plan that a patient cannot implement is not a good plan. Simplification improves compliance:

• Create a phased protocol: foundational lifestyle changes first, then layer hormones or medications as needed.
• Use habit loops and environmental cues.
• Consider delivery methods that improve adherence—transdermal patches, long-acting injectables, or, for some BHRT candidates, pellet therapy—while carefully weighing benefits, risks, and patient preference.

An example from my clinic: a perimenopausal patient with low adherence to oral medications achieved stable symptom control with transdermal estradiol and oral micronized progesterone, paired with a Mediterranean-pattern nutrition plan and short, structured resistance workouts. Adherence soared because the plan fit her life.

Objective Measurement: From Guesswork to Precision

We measure what matters:

• Metabolic: fasting glucose, insulin, HOMA-IR, HbA1c, lipid panel with ApoB, Lp(a), and triglyceride/HDL ratio.
• Inflammatory: hs-CRP, ferritin (contextualized), selected cytokines in research settings.
• Endocrine: TSH, free T4, free T3, estradiol, progesterone, testosterone (total and free), SHBG, DHEA-S, cortisol (clinical context).
• Nutritional: vitamin D, B12, folate, iron studies, magnesium (RBC if available), and omega-3 index.
• Functional: VO2 estimates, grip strength, gait speed, HRV, sleep metrics.

This data allows us to:

• Stratify risk and personalize intervention.
• Demonstrate progress and maintain engagement.
• Adjust therapy responsively and safely.

Clinical Vignettes from My Practice

1. Refractory Migraines

• • Findings: perimenopausal estrogen volatility; low magnesium; cervical myofascial dysfunction; sleep restriction.
  • Interventions: transdermal estradiol and micronized progesterone; magnesium glycinate; riboflavin; cervical mobilization and stabilization; CBT-I.
  • Outcome: >70% reduction in frequency and intensity within 12 weeks; lower reliance on abortive medications.

2. Resistant Hypertension with Metabolic Syndrome

• • Findings: high fasting insulin; elevated ApoB; poor sleep; high sympathetic tone.
  • Interventions: Mediterranean-pattern nutrition; strength plus Zone 2 training; time-restricted eating aligned with circadian rhythm; low-dose ARB; magnesium; stress reduction.
  • Outcome: BP normalized; 10% weight reduction; improved ApoB and HOMA-IR; medication de-escalation considered at 24 weeks.

3. Chronic Low Back Pain Post-Collision

• • Findings: paraspinal deconditioning; neuropathic features; sedentary behavior; Vitamin D deficiency; poor sleep.
  • Interventions: spine-neutral stabilization; graded motor imagery; anti-inflammatory nutrition; vitamin D repletion; sleep optimization; psychological resilience coaching.
  • Outcome: Pain decreased from 7/10 to 2/10; increased function and return to work; improved sleep quality.

These cases underscore a principle: integrated, personalized care achieves outcomes that monotherapy rarely matches.

The Economics and Ethics of Care

Healthcare costs explode when we ignore primary prevention and lifestyle medicine. Ethically, we must offer patients interventions with the best long-term risk-benefit profile and empower them to choose—backed by clear data and transparent discussion. Profit and purpose can coexist when outcomes drive decisions.

Education and Community: Practitioners as Well-Care Providers

We must redefine ourselves as well-care providers—not only disease managers. That requires:

• Ongoing education in nutrition, endocrinology, metabolism, and behavior change.
• Community among practitioners to share protocols, outcomes, and research updates.
• Partnerships that respect clinician judgment and patient autonomy.

When I consult with patients, I remember the “white coat power”: people still look to us for direction. Our responsibility is to guide with science and empathy.

Implementation Blueprint: From Clinic Vision to Daily Practice

Phase 1: Foundation”

• Nutrition: personalize macronutrient distribution; eliminate ultra-processed foods; increase fiber and omega-3 intake.
• Movement: schedule strength and Zone 2 sessions; track step count and HRV.
• Sleep: establish consistent schedules; implement CBT-I for insomnia.
• Stress: teach daily breathing and brief mindfulness routines.

Phase 2: Endocrine Precision

• Address thyroid disorders; consider BHRT for appropriate candidates using bioidentical hormones; treat hypogonadism judiciously.
• Monitor labs and symptoms; titrate carefully.

Phase 3: Targeted Adjuncts

• Address micronutrient deficiencies.
• Consider pharmacologic agents (e.g., metformin, GLP-1 receptor agonists) when lifestyle alone is insufficient, and the benefits outweigh the risks.
• Evaluate peptide or biologic options within regulatory and evidence boundaries.

Phase 4: Sustain and Iterate

• Quarterly or semiannual check-ins; adjust based on labs and life changes.
• Maintain core lifestyle practices; update goals and training plans.

Safety, Monitoring, and Informed Consent

Every therapy—lifestyle, supplement, hormone, peptide, or drug—requires:

• Clear indications and alternatives.
• Baseline and follow-up labs or metrics.
• Risk counseling and informed consent.
• Adverse effect surveillance and deprescribing when appropriate.

Primum non nocere is not passive—it is active diligence.

Research Methods and Evidence Integration

I emphasize:

• Systematic reviews and meta-analyses for the scope.
• Randomized controlled trials for causality (with thoughtful external validity appraisal).
• Cohort and case-control studies for real-world insights.
• N-of-1 trials in individualized care when appropriate.

We interpret data through the lens of mechanisms: mitochondrial biology, endocrine feedback loops, immunometabolism, and circadian physiology. This mechanistic triangulation reduces the likelihood of chasing correlations and enhances clinical relevance.

Toward a Unified Model: Science, Humanity, and Critical Thinking

When science and humanity converge, care becomes both art and evidence. We must teach patients how their bodies work—how insulin, cortisol, estrogen, and thyroid hormones interact, how sleep and stress shape metabolism, how nutrition sculpts the microbiome and immune tone. When patients understand, adherence becomes self-advocacy.

Summary

Creation-to-summary timeline: This post was created on 2026-01-16 07:40:15. The following summary condenses the concepts presented up to this date.

Summary

• The future of medicine is integrative and personalized, grounded in modern evidence and root-cause reasoning.
• Symptom suppression alone perpetuates chronic disease. Foundational therapies—nutrition, movement, sleep, and stress regulation—must precede or accompany medications and procedures.
• Hormone optimization (bioidentical estrogen, progesterone, testosterone, thyroid hormone) has a place when clinically indicated and carefully monitored. Benefits include improved quality of life, metabolic resilience, and tissue health.
• Lipid management should consider ApoB and particle number, not just LDL-C. Cardiovascular risk-modification pairs best with lifestyle redesign.
• Medical freedom—patient choice and clinician autonomy—is integral to quality. Evidence-based integrative therapies deserve equal consideration.
• Adherence is an outcome. Simplify plans and use delivery systems that support real-world implementation.
• Objective measurement (metabolic, inflammatory, endocrine, functional) enables precision and accountability.
• Pragmatic protocols and continuous education empower providers to become well-care practitioners.

Conclusion

From my perspective as a DC and APRN, the path forward is clear: blend rigorous science with humane, individualized care. Our responsibility is to ask “why,” to trace symptoms back to the physiology sustaining them, and to intervene at the most causal level possible. When we do, patients get better—often faster and more completely than with reactive strategies alone. The future is proactive healthcare: empowered patients, informed clinicians, and outcomes that testify to the value of combining lifestyle medicine with judicious pharmacology and precise endocrinology.

Key Insights

• Root-cause frameworks outperform symptom-only approaches in chronic disease.
• Nutrition, sleep, movement, and stress regulation are high-yield, evidence-backed therapies.
• Hormone therapy, when indicated and monitored, can restore function and quality of life.
• Personalized lipid management should assess particle burden and inflammatory context.
• Clinical autonomy and patient choice are ethical imperatives tied to the quality of outcomes.
• Measurement drives mastery: track what matters, iterate based on data.
• Simplicity scales: the best plans are the ones patients can live with—consistently.

References:

• American College of Cardiology/American Heart Association guidelines on lipid management and prevention.
• North American Menopause Society position statements on hormone therapy.
• Endocrine Society guidelines on testosterone therapy in men with hypogonadism.
• USPSTF recommendations for lifestyle interventions in cardiometabolic risk.
• Systematic reviews on Mediterranean diet and cardiovascular outcomes.
• Evidence for CBT-I as first-line therapy for chronic insomnia.
• Reviews on immunometabolism, mitochondrial biogenesis (PGC-1?), and exercise physiology.
• Literature on ApoB and atherosclerotic cardiovascular risk stratification.

Keywords:

• Integrative Medicine, Functional Medicine, Personalized Medicine, Dr. Alexander Jimenez, Root Cause Healing, Hormone Optimization, Bioidentical Hormone Replacement Therapy (BHRT), Pellet Therapy, Thyroid Function, Nutrition, Medical Freedom, Cholesterol, Statin Drugs, Alzheimer’s Disease, Dendritic Cells, Immune Function, Women’s Health Initiative (WHI), Evidence-Based Medicine, Proactive Healthcare, Sick Care vs. Well Care, Practitioner Education

Disclaimers:

• This educational post by Dr. Alexander Jimenez, DC, APRN, FNP-BC, is for informational purposes only and should not be used as medical advice.
• All individuals must obtain recommendations for their personal situations from their own licensed medical providers.